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Title:	ECG in Primary Care: Beyond the Basics – Improving Your 12-Lead ECG Interpretation Skills
Dates:	January 31, 2022 - February 3, 2022
Location:	Hyatt Regency, Sarasota, FL
Daily Schedule:	7:30am-12:50pm EST
Course #:	SEMLA-3620220131
Type:	In-Person & Live Webinar

Agenda & Learning Objectives

1 Day

Methodical Approach to ECG Interpretation Upon completion of this session, the participant should be able to: ^COMP

Recognize the importance of a fixed, methodical approach to 12-lead ECG interpretation.
Demonstrate the recommended order of 12-lead ECG analysis.
Utilize the “Three Main Causes” approach to abnormalities encountered while interpreting a 12-lead ECG.
Integrate participant’s own adjustments to the methodical approach without reducing sensitivity.

Normal/Abnormal P Waves, PR Intervals and QRS Complexes Upon completion of this session, the participant should be able to: ^COMP

Distinguish P waves originating outside the SA node.
Differentiate PR intervals that indicate AV conduction from those that do not.
Appraise the appropriateness of the QRS intervals in relation to the leads in which they appear.
Distinguish normal septal q waves from pathological Q waves.

Normal/Abnormal ST Segments and T Waves Upon completion of this session, the participant should be able to: ^COMP

Distinguish between normal ST segments and ST segments that deviate abnormally from the baseline.
Relate the different appearances of an abnormal ST segment.
Detect abnormalities in the T wave.
Appraise the QTc interval for abnormal durations.

Acquiring the ECG: Lead Switches, Axes, Loose Electrodes Upon completion of this session, the participant should be able to: ^COMP

Determine the Mean QRS Axis (ÂQRS) in the frontal plane.
Relate why a particular deflection may be positive in one lead but negative in another.
Utilize the lead axes to determine if a lead wire switch has occurred.
Employ the hexaxial grid to determine the location of a faulty electrode application.

The 12 Leads: What Do They See? How Do I Group Them? Upon completion of this session, the participant should be able to: ^COMP

Analyze the 12-lead ECG in terms of inferior, anterior, posterolateral, anteroseptal and anterolateral patterns.
Use the knowledge of Einthoven’s Triangle to determine the source of an electrode connectivity artifact.
Apply knowledge of correct deflection morphologies in detecting the presence of lead wire interchanges.

2 Day

Bundle Branch Block Upon completion of this session, the participant should be able to: ^COMP

Distinguish the classic morphologies of complete right bundle branch block (cRBBB) and complete left bundle branch block (cLBBB).
Differentiate the repolarization abnormality from acute ischemia.
Distinguish between true complete bundle branch block and ventricular ectopy.
Distinguish nonspecific interventricular conduction defects from classic bundle branch block.

AV Block: 1st, 2nd and 3rd Degree AV Block Upon completion of this session, the participant should be able to: ^COMP

Interpret a 1st Degree AV block in relation to the patient’s age and physical condition.
Detect a Mobitz Type I 2nd degree AV block and relate it to a cause, a prognosis and a treatment.
Detect a Mobitz II 2nd degree AV block and relate it to a cause, a prognosis and a treatment.
Differentiate Mobitz Type I and Mobitz Type II AV blocks with regards to the site of block.

ECG Recognition and Practice: AV Blocks and AV Dissociation Upon completion of this session, the participant should be able to: ^COMP

Distinguish a 3rd degree AV block from a simple AV dissociation.
Discuss capture beats and fusion beats and demonstrate their utility in the diagnosis of AV dissociation and its differentiation from 3rd degree AV block.
Relate the differences between a junctional escape pacemaker and a ventricular escape pacemaker.
Illustrate the differences between a 3rd degree AV block in the AV node and one in the infranodal regions.

Chamber Enlargement – LVH and the Confusion It Causes Upon completion of this session, the participant should be able to: ^COMP

Relate at least two different methods of determining left ventricular hypertrophy.
Detect the electrocardiographic signs of right ventricular hypertrophy.
Distinguish right atrial abnormality from left atrial abnormality.
Relate the presence or absence of a repolarization abnormality in RVH and LVH to patient prognosis.

Detecting Potentially Lethal Conditions Before Disaster Strikes Upon completion of this session, the participant should be able to: ^{GL, COMP}

Detect the ECG changes of Brugada Syndrome.
Detect changes suggestive of hyperkalemia.
Detect the changes of Wellens Syndrome.
Detect de Winter T wave changes and relate their importance.

3 Day

Atrial Tachycardia, Atrial Fibrillation and Atrial Flutter Upon completion of this session, the participant should be able to: ^COMP

Distinguish atrial tachycardia from sinus tachycardia.
Distinguish atrial flutter from both atrial tachycardia and sinus tachycardia.
Distinguish sinus tachycardia from atrial flutter with 2:1 conduction.
Detect 3rd degree AV block in the presence of atrial fibrillation.

The AV Tachycardias: AVNRT and AVRT Upon completion of this session, the participant should be able to: ^COMP

Relate the typical P wave changes in AVNRT relative to the QRS complex.
Relate the mechanism of AVNRT and how that knowledge is used to terminate the dysrhythmia.
Differentiate between an AVNRT and an AVRT.
Relate the inherent dangers of an accessory pathway.

Wide Complex Tachycardias (WCT) | Ventricular Tachycardia Upon completion of this session, the participant should be able to: ^COMP

Distinguish between classic bundle branch block morphology and ectopy.
Utilize three “quick signs” that are highly suggestive of ventricular tachycardia as the cause of a WCT.
Relate and utilize the Brugada Algorithm in diagnosing ventricular tachycardia.
Assess a WCT for its differential diagnosis of four possibilities: ventricular tachycardia, SVT with aberrancy, antidromic AVRT and “toxic tachycardia” associated with hyperkalemia or Na+ channel blocker toxicity.

ECG Recognition and Practice: Tachycardias Upon completion of this session, the participant should be able to: ^{GL, COMP}

Demonstrate the ability to utilize the Methodical Approach to 12-lead ECG interpretation while incorporating the “Three Main Causes” approach to assessing any abnormalities encountered during the interpretation.
Demonstrate the ability to recognize the different forms of AV block and to differentiate 3rd degree AV block from simple AV dissociation.
Specify the classic morphological features of left and right bundle branch block and the importance of the repolarization abnormality.
Recognize the main categories of subendocardial and acute epicardial ischemia.
Relate the importance and use of reciprocal changes in diagnosing acute epicardial ischemia.

5 Things You MUST Look For Before Putting Down an ECG | 5 Fatal Mistakes To AVOID While Interpreting an ECG Upon completion of this session, the participant should be able to: ^COMP

List 5 conditions for which the ECG must be specifically and carefully scrutinized.
Specify 5 analytical or interpretative mistakes that could result in increased patient morbidity or fatality.
Integrate steps to avoid missing important signs on the 12-lead ECG into the methodical approach.

4 Day

Ischemia: Subendocardial, Epicardial, Hyperacute T’s, Jones’s Sign (Jones’s Rule) Upon completion of this session, the participant should be able to: ^COMP

Differentiate between subendocardial ischemia and epicardial ischemia.
Analyze a 12-lead ECG for the earliest signs of myocardial ischemia.
Detect “Jones’s Sign” and assess minimal ST changes as possible early signs of ischemia.
Distinguish between inferior, anteroseptal (anteroapical), anterolateral and posterolateral epicardial ischemias.

Reciprocal changes: What are they and how do they help? | Timeline for ST – T Resolution Post-MI Upon completion of this session, the participant should be able to: ^COMP

Differentiate between ST depression as an indication of subendocardial ischemia or as a reciprocal change.
Utilize serial 12-lead ECGs to detect subtle changes in the ST segment when the initial ECG is non-diagnostic.
Detect dysrhythmias characteristic and diagnostic of reperfusion.
Relate current ST deviation in relation to the elapsed time following reperfusion.

Acute Coronary Syndromes with ST Elevation: Inferior, Anterior, Basolateral, Posterolateral Upon completion of this session, the participant should be able to: ^COMP

Detect ST elevations indicating acute inferior, anterior, basolateral and posterolateral epicardial ischemia.
Distinguish between anterior subendocardial ischemia and posterolateral epicardial ischemia.
Recognize signs of a very proximal LAD occlusion causing anterior ischemia.
Recommend a preferred approach to a patient with chest pain and a non-diagnostic admission 12-lead ECG.

ECG Recognition and Practice: ST Elevation ACS Syndromes Upon completion of this session, the participant should be able to: ^COMP

Distinguish between ischemic ST elevation and early repolarization.
Recognize the subtleties of Lead aVL during acute epicardial ischemia.
Employ the presence of reciprocal changes in the diagnosis of an acute MI.
Distinguish between subendocardial ischemia and reciprocal changes.

General

Formulate an approach to analyze a dysrhythmia.

Recognize the common AV blocks and state which causes may be present and what complications to anticipate.

Decide which patients must be treated PRIOR to obtaining a diagnosis.

Differentiate between the two most common narrow-complex tachycardias and know which one could be potentially lethal.

Accreditations

AMA
American Medical Seminars, Inc. designates this live activity for a maximum of 20 AMA PRA Category 1 Credits™. Physicians should claim only the credit commensurate with the extent of their participation in the activity.

Venue

Hyatt Regency, Sarasota, FL

1000 Boulevard of the Arts
Sarasota, FL 34236 United States + Google Map

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Group Discount Code – Corp #XZ35AMS

Presented By

Jerry W. Jones, MD

Meet Dr. Jerry W. Jones

Board-certified emergency physician, author, speaker, instructor and and internationally-recognized expert in electrocardiography.

Medicus of Houston was established by Jerry W. Jones, MD – a diplomate of the American Board of Emergency Medicine who has practiced internal medicine and emergency medicine for over 40 years. Dr. Jones is a graduate of Tulane University and the University of Texas Health Science Center at San Antonio. After a residency in internal medicine, he became one of the first full-time emergency physicians – even before emergency medicine residencies and board certification.

Dr. Jones has been on the teaching faculties of the University of Oklahoma and The University of Texas Medical Branch in Galveston. He is a published author who has also been featured in the New York Times and the Annals of Emergency Medicine for his work in the developing field of telemedicine. In addition, Dr. Jones is also a Fellow of the American College of Emergency Physicians, a Fellow of the American Academy of Emergency Medicine and a member of the European Society of Emergency Medicine.

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